This activity is expired and is no longer available for CME credit.


Nitric Oxide Signaling and Its Effect on Outflow Pathways in Glaucoma

Author(s)/Faculty: Inder Paul Singh, MD; Jason Bacharach, MD; Nathan M. Radcliffe, MD
Release Date: 3/8/2019Expiration Date: 2/26/2021
Credit Type: CMENumber of Credits: 0
Content Type: LectureProvider:
Glaucoma is a leading cause of blindness worldwide, with an estimated 70 million people living with this condition. Many individuals with glaucoma remain at risk for disease progression due to inadequate control of intraocular pressure (IOP). Studies have demonstrated that nitric oxide (NO) plays a role in the regulation of IOP through the potential modification of trabecular outflow facility, identifying a new target for IOP control. In this educational activity, experts in the field will discuss the role of NO in IOP reduction, and review clinical studies assessing NO donating therapies in the treatment of glaucoma. The presentation of challenging clinical cases will further provide guidance for the integration of NO donating therapies into practice.